What causes post traumatic iridodonesis?

What causes iridodonesis? The primary cause of iridodonesis is the subluxation of the lens. This may occur as a result of glaucoma, cataract formation, post-surgical complications, or trauma to the eye.

What is Neuroadaptation after cataract surgery?

A process of neuroadaptation, the capability of the brain to adapt to changes, can be activated after Mu-IOL implantation to adjust the neurophysiology of the changes that are induced in the quality of the retinal image by light dispersion.

How long does traumatic mydriasis last?

The dilation from mydriatics gradually wears off over four to eight hours. In some instances, it may last as long as 24 hours, however. While your eyes are dilated, you’ll have temporary eye sensitivity to bright light. Mydriatics rarely have other effects on the body.

Can posterior capsular opacification go away by itself?

Can PCO come back? You will generally only need to have laser treatment for PCO once. Very rarely, certain cells may develop in the area of the posterior capsule, or the opening in the capsule made by the first laser treatment can shrink, causing PCO to come back.

Is posterior capsule opacification an emergency?

What happens if PCO is not treated? PCO is not a serious disease nor is it an emergency. If it is not treated, it will increase with time and cause deterioration of vision.

What is Dysphotopsia?

dysphotopsia—positive and negative—that can occur after cataract surgery. “Dysphotopsia just means an unwanted image that patients see after cataract surgery.

How long does it take for the brain to adjust to cataract surgery?

In order to fully appreciate the benefit of cataract surgery, it is often necessary to wait for your eyes and brain to adjust to your new lenses which may require one or two weeks.

Is lens subluxation an emergency?

Extraction of a subluxated or dislocated cataractous lens should be considered only when the cataract is causing significant visual impairment. Urgent intervention is indicated for cases of pupillary block glaucoma, intractable uveitis, or lens–corneal touch leading to corneal decompensation.